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By M. Chen, T. Pope, D. Ott(eds0
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Extra resources for Basic Radiology
In fact, in some circumstances, it is not necessary to perform another test because of the limited potential yield from the examination or because there is no adequate therapy for the suspected abnormality. It is hoped that future recommendations for test selection will be determined by well-designed prospective unbiased outcome studies comparing all of these modalities in various clinical scenarios. In the meantime, a commonsense approach, taking into consideration the history and physical examination findings, the information gleaned from the conventional radiograph, and the potential yield from the array of other available imaging tests, is the most appropriate tact.
B. tricuspid valve. C. aortic valve. D. pulmonary embolus. E. pericardial calcification. 3-22. In Case 3-22 (Fig. 3â 55B ) the arrows are pointing to A. pulmonary artery calcification. B. pericardial effusion. C. pericardial calcification. D. ascending aortic aneurysm. E. descending thoracic aortic aneurysm. 3-23. In Case 3-23 (Fig. 3â 56) the arrows on the lateral chest radiographs point to calcifications within which cardiac structure? A. Pericardium B. Mitral valve C. Aortic valve 57 58 D. Tricuspid valve E.
Right lung hypoplasia causes the small size of the right hemithorax and results in shift of the heart and mediastinum to the right. Other congenital anomalies may be present. Fig. 3â 45. Axial CT with contrast shows anomalous right pulmonary vein descending (*) and entering (arrow) inferior vena cava. AZ = azygous vein. Pulmonary edema, as exhibited in Case 3-15 (Fig. 3â 44), regardless of the cause, is another process that causes the increase in the pulmonary vascularity seen on chest radiograph (discussed further in the next chapter).